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Hope for Alzheimer's: Drug Reverses Cognitive Damage

Auburn researchers discover that troriluzole reduces harmful glutamate and improves memory in Alzheimer's models, paving the way for new treatments.

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Written by: Alejandra Legarda - Journalist

Recent studies on the drug troriluzole have shown promising results in treating Alzheimer's by preventing early brain changes related to memory loss and cognitive decline. Researchers at Auburn University discovered that troriluzole reduces harmful glutamate levels, which are associated with neural hyperactivity in the early stages of Alzheimer's. This reduction not only preserves synaptic function but also improves memory and learning in a mouse model designed to replicate the early stages of Alzheimer's.

Similar research, published in the Journal of Neurochemistry, highlights that troriluzole targets presynaptic alterations and could slow the progression of the disease by restoring synaptic and cognitive functions. It has the potential to offer new hope for patients, especially if proven effective in humans.

These findings align with other Alzheimer's research focusing on reducing neural hyperactivity caused by excess glutamate. Further studies are needed to explore how troriluzole may impact different stages of the disease, with the possibility of offering a preventive treatment.

In conclusion, the collaborative effort of Auburn University researchers emphasizes the potential of troriluzole as an innovative treatment for Alzheimer's, particularly if future research confirms its effectiveness in humans.

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